Imagine your body as a giant school. 

Most cells graduate or retire when their job is done. 

But some refuse to leave🤨

they just sit around, grumpy and useless, shouting angry messages that stress out the others. 

These are senescent cells — aka cellular zombies 😈

They don’t divide, don’t help, and worst of all, 

they spread inflammation and chaos. 

Over time, these zombie cells:

⚠️ Damage nearby tissues

⚠️ Slow down healing

⚠️ Speed up aging in skin, joints, organs — everywhere!

Here’s where Quercetin comes in — one of the key ingredients in AmCELL.

Quercetin: The Zombie Bouncer

Works like a senolytic agent,

it clears out zombie cells by switching on the “self-destruct” button.

Human studies show that just 11 days of treatment can reduce zombie cell markers in tissues.

It also acts as a senomorphic, helping calm down the bad messages even when some zombie cells stick around.

That’s like Quercetin walking into a zombie cell party with a megaphone:📣

“Party’s over!”

and half of them leave, while the rest finally shut up.

By keeping these troublemakers in check, Quercetin helps:

✅ reduce inflammation

✅ boost energy

✅ slow down the aging process.

🔍 Coming up: What are telomeres, and why are they like the plastic tips of your DNA shoelaces? 

Find out in Part 3 — before they fray away!

References:

Chondrogianni, N., Kapeta, S., Chinou, I., Vassilatou, K., Papassideri, I., & Gonos, E. S. (2010). Anti-ageing and rejuvenating effects of quercetin. Experimental gerontology, 45(10), 763-771.

Hickson, L. J., Prata, L. G. L., Bobart, S. A., Evans, T. K., Giorgadze, N., Hashmi, S. K., ... & Kirkland, J. L. (2019). Senolytics decrease senescent cells in humans: Preliminary report from a clinical trial of Dasatinib plus Quercetin in individuals with diabetic kidney disease. EBioMedicine, 47, 446-456.